Logo-apb
Adv Pharm Bull. 2023;13(4): 747-760.
doi: 10.34172/apb.2023.077
PMID: 38022805
PMCID: PMC10676553
  Abstract View: 989
  PDF Download: 624

Research Article

Targeted Delivery of Pennyroyal via Methotrexate Functionalized PEGylated Nanostructured Lipid Carriers into Breast Cancer Cells; A Multiple Pathways Apoptosis Activator

Amin Mahoutforoush 1,2,3 ORCID logo, Leila Asadollahi 2 ORCID logo, Hamed Hamishehkar 2* ORCID logo, Soheil Abbaspour-Ravasjani 2* ORCID logo, Atefeh Solouk 3 ORCID logo, Masoumeh Haghbin Nazarpak 4 ORCID logo

1 Immunology Research Center and Students Research Committee, Tabriz University of Medical Sciences, Tabriz, Iran.
2 Drug Applied Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
3 Biomedical Engineering Department, Amirkabir University of Technology (Tehran Polytechnic), Tehran 1591634311, Iran.
4 New Technologies Research Center (NTRC), Amirkabir University of Technology, Tehran 1591634653, Iran.
*Corresponding Authors: Hamed Hamishehkar, Email: Hamishehkarh@tbzmed.ac.ir; Soheil Abbaspour-Ravasjani, Email: Abbaspour.s@tbzmed.ac.ir

Abstract

Purpose: Pennyroyal is a species of the Lamiaceae family with potent anti-cancer and antioxidant properties. Combining this antioxidant with chemotherapeutic agents enhances the effectiveness of these agents by inducing more apoptosis in cancerous cells.

Methods: Here, methotrexate (MTX) combined with pennyroyal oil based on PEGylated nanostructured lipid carriers (NLCs) was assessed. These nanoparticles were physiochemically characterized, and their anti-cancer effects and targeting efficiency were investigated on the folate receptor-positive human breast cancer cell line (MCF-7) and negative human alveolar basal epithelial cells (A549).

Results: Results showed a mean size of 97.4±12.1 nm for non-targeted PEGylated NLCs and 220.4±11.4 nm for targeted PEGylated NLCs, with an almost small size distribution assessed by TEM imaging. Furthermore, in vitro molecular anti-cancer activity investigations showed that pennyroyal-NLCs and pennyroyal-NLCs/MTX activate the apoptosis and autophagy pathway by changing their related mRNA expression levels. Furthermore, in vitro cellular studies showed that these changes in the level of gene expression could lead to a rise in apoptosis rate from 15.6±8.1 to 25.0±3.2 (P<0.05) for the MCF-7 cells treated with pennyroyal-NLCs and pennyroyal-NLCs/MTX, respectively. Autophagy and reactive oxygen species (ROS) cellular evaluation indicated that treating the cells with pennyroyal-NLCs and pennyroyal-NLCs/MTX could significantly increase their intensity in these cells.

Conclusion: Our results present a new NLCs-based approach to enhance the delivery of pennyroyal and MTX to cancerous breast tissues.

First Name
Last Name
Email Address
Comments
Security code


Abstract View: 990

Your browser does not support the canvas element.


PDF Download: 624

Your browser does not support the canvas element.

Submitted: 30 Jul 2022
Revision: 26 Feb 2023
Accepted: 24 Apr 2023
ePublished: 29 Apr 2023
EndNote EndNote

(Enw Format - Win & Mac)

BibTeX BibTeX

(Bib Format - Win & Mac)

Bookends Bookends

(Ris Format - Mac only)

EasyBib EasyBib

(Ris Format - Win & Mac)

Medlars Medlars

(Txt Format - Win & Mac)

Mendeley Web Mendeley Web
Mendeley Mendeley

(Ris Format - Win & Mac)

Papers Papers

(Ris Format - Win & Mac)

ProCite ProCite

(Ris Format - Win & Mac)

Reference Manager Reference Manager

(Ris Format - Win only)

Refworks Refworks

(Refworks Format - Win & Mac)

Zotero Zotero

(Ris Format - Firefox Plugin)